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OXIDATION BEHAVIORS OF THE PHOTOVOLTAIC RIBBON IN THE SPENT PHOTOVOLTAIC MODULE

Published In: 6TH INTERNATIONAL CONFERENCE ON ADVANCES IN APPLIED SCIENCE AND ENVIRONMENTAL ENGINEERING
Author(s): JEI-PIL WANG , SAE-KWON HEO , SEOUNG-SIK JEOUNG

Abstract: The solar cell has been in the spotlight as new renewable energy. Considering its life expectancy, a large quantity of the spent module will be produced after 2020, so many studies are being conducted on the recovery of valuable resources from the spent photovoltaic module. In particular, there are not many studied conducted on the photovoltaic robbin recovered from the spent photovoltaic module. Therefore, in this study, experiments were conducted to verify the oxidation behaviors in the coating layer of the photovoltaic ribbon recovered from the spent photovoltaic module. The oxidation behaviors were observed for 24 hours at the temperature of 100°C, 300°C, 500°C and 700°C using TGA, and the oxide layer formation and the component change in respective sections were analyzed using SEM and EDC according to the holding time of 1hr, 2hrs, 4hrs and 8hrs at the temperature of 500°C showing the most distinctive oxidation behavior.

  • Publication Date: 27-Nov-2016
  • DOI: 10.15224/978-1-63248-108-5-36
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INITIAL STUDY OF THE MUTATION IN EXON 4 OF THE LDLR GENE IN VIETNAMESE PATIENTS WITH FAMILIAL HYPERCHOLESTEROLEMIA

Published In: 6TH INTERNATIONAL CONFERENCE ON ADVANCES IN APPLIED SCIENCE AND ENVIRONMENTAL ENGINEERING
Author(s): LAO DUC THUAN , LE HUYEN AI THUY , TRUONG KIM PHUONG

Abstract: Familial hypercholesterolemia (FH) has been identified as the genetic disorder disease, which is characterized by high total cholesterol, in particularly, the high level of low density lipoprotein (LDL). The normal function of low density lipoprotein receptor is obligate for the normal of lipid profile. The mutation in exon 4 of LDLR gene has been identified to be responsible to FH. In current study, the PCR sequencing method was applied to analyze the mutation of exon 4 of LDLR gene of forty clinical samples, which were diagnosed with FH. As the results, 29 samples of 40 samples, counting for 72.5%. Notably, the genotypes were heterogeneous. In the representative sample DA1, the mutation 355del and c.379G>T (p.V106F) were found, in which, the mutation c.379G>T (p.V106F) has not been previously reported previously. In conclusion, this study being the first comprehensive mutation analysis of the LDLR causing FH in our country. Additionally, mutations identification in exon 4 of LDLR gene

  • Publication Date: 27-Nov-2016
  • DOI: 10.15224/978-1-63248-108-5-41
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